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Mechanistically, microecological imbalance may weaken immune receptivity during the implantation window by activating Toll-like receptor signaling, inducing IL-1β-mediated local inflammatory responses, or interfering with the homeostatic function of uterine NK cells
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LPS itself can induce the production of other inflammatory cytokines, including interferon-gamma (IFN-γ) and interleukin-1 beta (IL-1β) (Garde et al., 1995)
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